Herpesviridaeinfections and urticaria were the most frequently reported associations in children.
2
Genetic studies have provided evidence of distinct immunopathological mechanisms and inherited susceptibility to IRD associated with mycobacterial and herpesviridaeinfections.
1
Patients receiving single-agent fludarabine had more major infections and herpesvirusinfections compared with chlorambucil-treated patients.
1
Genetic studies have provided evidence of distinct immunopathological mechanisms and inherited susceptibility to IRD associated with mycobacterial and herpesviridaeinfections.
2
Herpesviridaeinfections and urticaria were the most frequently reported associations in children.
Uso de herpesvirus infection en inglés
1
Endothelial cells are important targets for herpesvirusinfection.
2
Background: Lymphoma after heart transplantation (HT) has been associated with induction therapy and herpesvirusinfection.
3
Both gels prevented genital herpesvirusinfection in mice, even when virus was introduced in human seminal plasma.
4
KEGG pathway analysis of specifically changed DEUs in respective clones revealed distinct immune responses to the herpesvirusinfection.
5
Previous studies identified a role for latent herpesvirusinfection in cross-protection against infection and exacerbation of chronic inflammatory diseases.
6
Intriguingly, in vivo modeling has shown that herpesvirusinfection induces or worsens lung fibrosis when combined with immunodeficiency or other injurious stimuli.
7
Comprehensive analysis of the shared DEUs in all three clones displayed common defense pathways to the herpesvirusinfection, activating IFN system and suppressing complements.
8
Interfering with either of these steps in the herpesvirus life cycle may offer a novel strategy for controlling herpesvirusinfection and associated disease pathogenesis.
9
Patients receiving single-agent fludarabine had more major infections and herpesvirusinfections compared with chlorambucil-treated patients.
10
Herpesvirusinfection was associated with reduced naive CD8 T cell precursors above the loss attributable to aging.